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Association between blood Interleukin-10 level in coronary artery disease: A systematic review, meta-analysis, and network analyses

Reza Heidari Moghadam 1
Mohammad Rouzbahani 1
Nahid Salehi 1, *
Masoud Sadeghi 2
  1. Cardiovascular Research Center, Health Institute, Imam Ali Hospital, Kermanshah University of Medical Sciences, Kermanshah, Iran
  2. Medical Biology Research Center, Health Technology Institute, Kermanshah University of Medical Sciences, Kermanshah, Iran
Correspondence to: Nahid Salehi, Cardiovascular Research Center, Health Institute, Imam Ali Hospital, Kermanshah University of Medical Sciences, Kermanshah, Iran. Email: [email protected].
Volume & Issue: Vol. 11 No. 7 (2024) | Page No.: 6603-6621 | DOI: 10.15419/bmrat.v11i7.907
Published: 2024-07-31

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This article is published with open access by BioMedPress. This article is distributed under the terms of the Creative Commons Attribution License (CC-BY 4.0) which permits any use, distribution, and reproduction in any medium, provided the original author(s) and the source are credited. 

Abstract

Background: Cytokines can be key factors in the pathogenesis of coronary artery disease (CAD). This systematic review and meta-analysis aimed to assess the levels of interleukin-10 (IL-10), an anti-inflammatory cytokine, in the serum/plasma of patients with CAD.

Methods: An exhaustive search was conducted across the Web of Science, PubMed/Medline, Scopus, and Cochrane Library databases up to March 25, 2022. Review Manager 5.3 software was used to calculate the effect sizes, presenting the standardized mean difference (SMD) along with a 95% confidence interval (CI). STRING software, which maps protein-protein interactions (PPI), was utilized to explore the functional interactions among the genes under study.

Results: From the 1130 records retrieved from the databases, 26 articles were included in the meta-analysis. The pooled SMD for CAD cases compared to controls was 0.33 (p = 0.15). The sample size was adequate for comparing blood IL-10 levels in CAD patients versus controls.

Conclusion: The findings suggest there was no significant difference in the serum/plasma levels of IL-10 between CAD patients and controls. Hence, the pathogenesis of CAD can be multifactorial and complex.

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